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Methylene Blue is not meant for internal consumption and is intended for research purposes only.
Made in 1876, Methylene Blue (MB) became the first ever fully synthetic material to be used in medicine.
Initially, Methylene Blue was used as a treatment for malaria in WWII by Allied Forces, and for psychiatric disorders such as schizophrenia. But as the research continued, it was realized that MB has a seriously broad spectrum of action and benefit.
What does Methylene Blue actually do? Methylene Blue (MB) has the fantastic ability of being able to repair damaged tissue, cells and mitochondria, allowing them to restore proper energy function.
It restores the most proper, organized, and efficient pathway of energy production in the mitochondria, where oxygen and carbohydrate are consumed, and ATP, CO2, and water are created.
It specifically has its effect in the electron transport chain (ETC), where it can act as a redox agent, meaning it can reduce itself, or oxidize itself wherever necessary. This means that it can donate electrons in the ETC where more electrons are needed, and can receive electrons when there are too many. This is a large component of its reparative effect.
Methylene Blue is not meant for internal consumption and is intended for research purposes only.
Made in 1876, Methylene Blue (MB) became the first ever fully synthetic material to be used in medicine.
Initially, Methylene Blue was used as a treatment for malaria in WWII by Allied Forces, and for psychiatric disorders such as schizophrenia. But as the research continued, it was realized that MB has a seriously broad spectrum of action and benefit.
What does Methylene Blue actually do? Methylene Blue (MB) has the fantastic ability of being able to repair damaged tissue, cells and mitochondria, allowing them to restore proper energy function.
It restores the most proper, organized, and efficient pathway of energy production in the mitochondria, where oxygen and carbohydrate are consumed, and ATP, CO2, and water are created.
It specifically has its effect in the electron transport chain (ETC), where it can act as a redox agent, meaning it can reduce itself, or oxidize itself wherever necessary. This means that it can donate electrons in the ETC where more electrons are needed, and can receive electrons when there are too many. This is a large component of its reparative effect.
